Reductions in circulating endocannabinoid levels in individuals with post-traumatic stress disorder following exposure to the World Trade Center attacks.

Author(s): Hill MN, Bierer LM, Makotkine I, Golier JA, Galea S, McEwen BS, Hillard CJ, Yehuda R

The Hotchkiss Brain Institute, Department of Cell Biology & Anatomy and Psychiatry, University of Calgary, Calgary, AB, Canada; Laboratory of Neuroendocrinology, The Rockefeller University, New York, NY, USA. Electronic address: [email protected]

Subject: PTSD View the study

Abstract:

Endocannabinoid (eCB) signaling has been identified as a modulator of adaptation to stress, and is integral to basal and stress-induced glucocorticoid regulation. Furthermore, interactions between eCBs and glucocorticoids have been shown to be necessary for the regulation of emotional memories, suggesting that eCB function may relate to the development of post-traumatic stress disorder (PTSD). To examine this, plasma eCBs were measured in a sample (n=46) drawn from a population-based cohort selected for physical proximity to the World Trade Center (WTC) at the time of the 9/11 attacks.

Source: Author Manuscript
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